Now in a study published in the journal PLOS ONE, Vsevolod Gurevich, PhD, and colleagues show that a glycine residue at the beginning of the finger loop is critical for the arrestin-GPCR interaction.
Researchers at Vanderbilt University Medical Center and colleagues have identified a gene that increases the risk for a severe and potentially life-threatening reaction to the commonly prescribed antibiotic vancomycin.
Danny Winder, PhD, and colleagues reported in The Journal of Neurosciencethat acute restraint stress in mice activates CRF neurons in the BNST, supporting a role for these neurons in stress-related behaviors.
Cellular signaling pathways involved in everything from the proliferation of fatty tissue to the death of neurons in the brain are tightly regulated by “cascades” of sequentially activated enzymes, MAP kinases.
